Bifidobacterium breve Bif195 Protects Against Small-intesti…

A CCEPTED MANUSCRIPT

response as well as compete with potential bacterial aggressors. The molecular details of

bifidobacterial-mediated protection against small-intestinal epithelial injury are currently under

investigation, but one candidate includes the pilus-associated protein Tad E which exerts a

proliferative effect on host colonic epithelium following oral consumption of B. breve 18 . This

appears to be a characteristic of all B. breve and supports our choice of the stain used in this trial.

Interestingly, fecal microbiome analysis revealed changes were limited to a marked increase in the

total B. breve population in the Bif195 arm. These data provide further evidence that microbial

intervention strategies targeting the microbiome can be clinically efficacious without inducing

major alterations in the overall microbial population structure.

Our 6-week ASA challenge model yielded minor responses in the GSRS questionnaire and in the

biomarkers of damage, I-FABP in blood and calprotectin in blood and feces. Although trends were

observed for I-FABP, only the fecal calprotectin endpoint reached statistical significance indicating

MANUSCRIPT

a modest Bif195 protective effect. Our data suggest that VCE is the method of choice when

conducting human challenges with mild induction of small-intestinal damage by NSAIDs over a

limited time period.

Although encouraging, the present clinical trial has limitations in terms of translation to a real-life

clinical setting. The relatively short-term challenge in healthy volunteers, for proof-of-concept, used

a higher dose of ASA than is most commonly prescribed for primary CVD prevention. However, it

is a dose that is readily available for over-the-counter usage. It is also noteworthy, that a recent

report suggested that the current cardioprotective dosage of ASA may be insufficient

and recommended doses based on a mg/kg basis 23 .

Due to our AUC approach based on a polynomic curve fitted to data-points obtained from 6

different visits, data imputation is not feasible for drop-out subjects where only baseline data are

available. Therefore, we acknowledge that long-term intervention clinical trials will be needed to

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